研究领域（方向）
心肌病、心衰；地方性心肌病（克山病）；心脏康复治疗
个人及工作简历
1988年于西安交通大学医学部本科毕业后，留校在二附院心内科工作至今，1999年获内科学博士学位（MD，PhD），2008年晋升主任医师，教授，并通过博士生导师遴选。
科研项目
[1] 国家“12.5”科技支撑项目横向课题，病毒性心肌炎演变扩张型心肌病的监测和防治策略研究，已结题，子课题负责人。
[2] 国家自然科学基金项目，Calreticulin-STAT3/PKC 信号通路介导的线粒体损伤在扩张型心肌病发病中的分子机制研究，已结题，主持。
[3]国家自然科学基金项目：PINK1/Parkin介导线粒体自噬参与病理性心肌肥厚的机制及其分子调控研究，在研，主持。
学术及科研成果、专利、论文
(1) Inhibition of drp1 attenuates mitochondrial damage and myocardial injury in coxsackievirus b3 induced myocarditis. Biochem Biophys Res Commun 2017： 550~556 484(3)
(2) Chronic resveratrol treatment improves cardiac function in a rat model of diabetic cardiomyopathy via attenuation of mitochondrial injury and myocardial apoptosis. Int J Clin Exp Med 2016 ，9(11)21156~21167
(3) Rosmarinic Acid suppressed high glucose-induced apoptosis in H9c2 cells by ameliorating the mitochondrial function and activating STAT3.Biochem Biophys Res Commun 2016， 477(4) : 1024~1030
(4) Involvement of caspases and their upstreamregulators in myocardial apoptosis in a rat model of endemic dilatedcardiomyopathy ，Journal of Trace Elements in Medicine and Biology 2015.7.1 31(2015) : 85~91
(5) Suppression of PKCε-medicated mitochondrial connexin 43 phosphorylation at serine 368 isinvolved in myocardial mitochondrial dysfunction in a rat model of dilated cardiomyopathy ，Molecular Medicine Reports 2015.3.6 11(6) : 4720~4726
(6) Impairment of myocardial mitochondria in viralmyocardial disease and its reflective window in peripheral cells，PLos One 2014.12.31 9(12) : 1~18
(7) Calreticulin is localized at mitochondria of rat cardiomyocytes and affected by furazolidone，Molecular and Cellular Biochemistry 2014.8.3 397(1-2) : 125~130
(8) Novel distribution of calreticulin to cardiomyocyte mitochondria and its increase in a rat model of dilated cardiomyopathy，Bioch emical and Biophysical Research Communications 2014.6.20 449(1) : 62~68
(9) Calreticulin-STAT3 signaling pathway modulates mitochondrialfunction in a rat model of furazolidone-induced dilated cardiomyopathy ，PLos One 2013.6.20 8(6)
(10) Selenium deficiency induces apoptosis through endoplasmic reticulum stress in a rat model of endemic dilated cardiomyopathy ，Experimental & clinical cardiology 2014.4.10 20(8) : 3076~3081
联系方式
联系电话：13572140118
电子邮箱：weijin@mail.xjtu.edu.cn
